Dacogen
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UNDERSTANDING MYELODYSPLASTIC SYNDROMES

Myelodysplastic syndromes (MDS) belong to a group of diseases that result from ineffective blood cell production.4 All blood cells are made in bone marrow. Even though all blood cells start out from stem cells, they eventually divide and mature into red blood cells, white blood cells or platelets.

In patients with MDS, this maturing process is damaged. Some stem cells become ineffective and do not mature into healthy cells. Instead, the ineffective stem cells produce defective cells. These cells live longer and outnumber the cells produced by healthy stem cells. The population of ineffective cells increases, while the population of healthy cells decreases.

Doctors who specialize in blood-related diseases consider MDS a "clonal" disease, which means many abnormal cells develop from one abnormal cell. Approximately 30% of patients with MDS will develop acute myelogenous leukemia (AML), a type of cancer that occurs in the bone marrow cells.

Learn more about Myelodysplastic Syndromes:

References

DACOGEN is indicated for treatment of patients with myelodysplastic syndromes (MDS), including previously treated and untreated, de novo, and secondary MDS of all FAB subtypes (RA, RARS, RAEB, RAEB-t, CMML) and Intermediate-1, Intermediate-2, and High-Risk IPSS groups.

DACOGEN may cause fetal harm when administered to a pregnant woman. Women of childbearing potential should be advised to avoid becoming pregnant while using DACOGEN. Men should be advised not to father a child while receiving treatment with DACOGEN, and for 2 months afterwards. The most commonly occurring adverse reactions include neutropenia, thrombocytopenia, anemia, fatigue, pyrexia, nausea, cough, petechiae, constipation, diarrhea, and hyperglycemia.

DACOGEN is available by prescription only. Please see the important product information for DACOGEN for more information. This site does not contain everything that is known about DACOGEN. If you would like to know more, talk to your healthcare professional.